Skip to main content
Home AIM
  • News
    All News Vibrant Campus Initiative Annual Meetings & Symposia Core Newsletters
  • Events
    All Events The AIM Center's 2021 eSymposia workshop
    Calendar
  • Cores
  • Research
    Funded Projects Publications
    Research and Funding Opportunities
  • People
  • How to acknowledge AIM
  • Log in

Metabolism

Fatty Acid Metabolites Combine with Reduced β Oxidation

Fatty Acid Metabolites Combine with Reduced β Oxidation to Activate Th17 Inflammation in Human Type 2 Diabetes

glycolysis
fatty acid oxidation
metaflammation
immunometabolism
Conserved functions in vertebrates

Roles of Regulatory T Cells in Tissue Pathophysiology and Metabolism

regulatory T cells
tissue homeostasis
Metabolism
CD8 T Cell glycolytic metabolism

Acylglycerol Kinase Maintains Metabolic State and Immune Responses of CD8+ T Cells

AGK
CD8+ T cells
glycolysis
PI3K-mTOR
Graphical Abstract

Epigenetic Control of Mitochondrial Fission Enables Self-Renewal of Stem-like Tumor Cells in Human Prostate Cancer

cancer stem cells
prostate cancer
mitochondrial dynamics
mitochondrial fission
UA phase 1 study

The mitophagy activator urolithin A is safe and induces a molecular signature of improved mitochondrial and cellular health in humans

Nature
mitophagy
urolithin a
mitochondria
Graphical abstract CD8+ T cell

Cholesterol Induces CD8+ T Cell Exhaustion in the Tumor Microenvironment

Cholesterol
cd8
t cell
immune checkpoints
Intestinal insulin/IGF1 signalling through FoxO1 regulates epithelial integrity and susceptibility to colon cancer

Intestinal insulin/IGF1 signalling through FoxO1 regulates epithelial integrity and susceptibility to colon cancer

Obesity
insulin
Colitis
Epithelial
Liver macrophages regulate systemic metabolism through non-inflammatory factors

Liver macrophages regulate systemic metabolism through non-inflammatory factors

liver
macrophage
metabolic
Secretion
Graphical abstract

Inhibiting Glutamine-Dependent mTORC1 Activation Ameliorates Liver Cancers Driven by β-Catenin Mutations

Wnt
beta-catenin
mTOR
glutamine
Graphical abstract of Pathogenic Th17 Cells

Calcium Signaling Controls Pathogenic Th17 Cell-Mediated Inflammation by Regulating Mitochondrial Function

pathogenic T helper 17 cells
Th17
IL-17
Colitis

Pagination

  • Previous page ‹‹
  • Page 2
Subscribe to Metabolism
Autophagy, Inflammation, & Metabolism Center of Biomedical Research Excellence

The AIM Center is funded by NIH grant P20GM121176, and it endows the state of New Mexico and the surrounding region with a state-of-the-art biomedical center and an intellectual and technological hub for cutting-edge research. On a larger scale, the AIM Center promises to be a nationally important center for the advancement of research on autophagy.

Information

  • About
  • Opportunities
  • FAQ
  • Privacy Policy

Contact Us

  • General Inquiries
  • Core Facilities & Services
  • Webmaster

©Copyright The AIM Center 2017-2022 |Sitemap