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mitochondria

Misregulation of mitochondrial 6mA promotes the propagation of mutant mtDNA and causes aging in C. Elegans

6mA
mtDNA
mitochondrial genome
mitochondria

Organization of a functional glycolytic metabolon on mitochondria for metabolic efficiency

mitochondria
glucose
glucose metabolism

Mitochondrial fatty acid synthesis is an emergent central regulator of mammalian oxidative metabolism

mitochondria
mitochondrial fatty acid synthesis
mtFAS
fatty acids

Metformin Enhances Autophagy and Normalizes Mitochondrial Function to Alleviate Aging-Associated Inflammation

inflammaging
T cells
metformin
Autophagy

Mitochondrial Integrity Regulated by Lipid Metabolism Is a Cell-Intrinsic Checkpoint for Treg Suppressive Function

mitochondria
Lipids
treg
UA phase 1 study

The mitophagy activator urolithin A is safe and induces a molecular signature of improved mitochondrial and cellular health in humans

Nature
mitophagy
urolithin a
mitochondria
Graphical abstract of Pathogenic Th17 Cells

Calcium Signaling Controls Pathogenic Th17 Cell-Mediated Inflammation by Regulating Mitochondrial Function

pathogenic T helper 17 cells
Th17
IL-17
Colitis
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Autophagy, Inflammation, & Metabolism Center of Biomedical Research Excellence

The AIM Center is funded by NIH grant P20GM121176, and it endows the state of New Mexico and the surrounding region with a state-of-the-art biomedical center and an intellectual and technological hub for cutting-edge research. On a larger scale, the AIM Center promises to be a nationally important center for the advancement of research on autophagy.

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