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mTORC1

Sustained activation of autophagy suppresses adipocyte maturation via a lipolysis-dependent mechanism.

Adipocyte maturation
mTORC1
Lipolysis
lysosome
Graphical abstract ATG9A

ATG9A shapes the forming autophagosome through Arfaptin 2 and phosphatidylinositol 4-kinase IIIβ

Autophagy
autophagosomes
mTORC1
ATG9A

Adipose mTORC1 Suppresses Prostaglandin Signaling and Beige Adipogenesis via the CRTC2-COX-2 Pathway.

COX-2
CRTC2
UCP1
adipose tissue
Adipose Research Full Image

Adipose mTORC1 Suppresses Prostaglandin Signaling and Beige Adipogenesis via the CRTC2-COX-2 Pathway

mTORC1
COX-2
CRTC2
UCP1
Subscribe to mTORC1
Autophagy, Inflammation, & Metabolism Center of Biomedical Research Excellence

The AIM Center is funded by NIH grant P20GM121176, and it endows the state of New Mexico and the surrounding region with a state-of-the-art biomedical center and an intellectual and technological hub for cutting-edge research. On a larger scale, the AIM Center promises to be a nationally important center for the advancement of research on autophagy.

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