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Metabolism

A Metabolic Handbook for the COVID-19 Pandemic

Metabolism
covid-19
sars

Metformin Enhances Autophagy and Normalizes Mitochondrial Function to Alleviate Aging-Associated Inflammation

inflammaging
T cells
metformin
Autophagy

Macrophage Metabolism of Apoptotic Cell-Derived Arginine Promotes Continual Efferocytosis and Resolution of Injury

Efferocytosis
arginine
ornithine
efferocytosis

High-protein diets increase cardiovascular risk by activating macrophage mTOR to suppress mitophagy

cardiovascular
macrophage
mTOR
mitophagy

Mitochondrial Integrity Regulated by Lipid Metabolism Is a Cell-Intrinsic Checkpoint for Treg Suppressive Function

mitochondria
Lipids
treg

Neutralization of Oxidized Phospholipids Ameliorates Non-alcoholic Steatohepatitis

Oxidize
phospholipid
Ameliorates
Steatohepatitis
Graphical Abstract

Pharmacological Activation of Pyruvate Kinase M2 Inhibits CD4+ T Cell Pathogenicity and Suppresses Autoimmunity

immunometabolism
PKM2
Th1
Th17
Graphical Abstract

STAT3 Activation-Induced Fatty Acid Oxidation in CD8+ T Effector Cells Is Critical for Obesity-Promoted Breast Tumor Growth

Obesity
FAO
PD-1
Leptin
Multidimensional mitophagy screen reveals that ANT is required for mitophagy

The ADP/ATP Translocase Drives Mitophagy Independent of Nucleotide Exchange

mitophagy
ant
nucleotide exchange
proteolysis

Lipid Signaling Drives Proteolytic Rewiring of Mitochondria by YME1L

mTOR
YME1L
proteolysis

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Autophagy, Inflammation, & Metabolism Center of Biomedical Research Excellence

The AIM Center is funded by NIH grant P20GM121176, and it endows the state of New Mexico and the surrounding region with a state-of-the-art biomedical center and an intellectual and technological hub for cutting-edge research. On a larger scale, the AIM Center promises to be a nationally important center for the advancement of research on autophagy.

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