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mTOR

High-protein diets increase cardiovascular risk by activating macrophage mTOR to suppress mitophagy

cardiovascular
macrophage
mTOR
mitophagy
proteolysis

Lipid Signaling Drives Proteolytic Rewiring of Mitochondria by YME1L

mTOR
YME1L
proteolysis
Graphical abstract

Inhibiting Glutamine-Dependent mTORC1 Activation Ameliorates Liver Cancers Driven by β-Catenin Mutations

Wnt
beta-catenin
mTOR
glutamine

Galectins Control mTOR in Response to Endomembrane Damage.

AMPK
APEX2
LC3
TAK1
Data Abstract

MITF-MIR211 axis is a novel autophagy amplifier system during cellular stress

Autophagy
cellular stress
lysosome
microRNA
Jingyue Gia - Abstract

Jingyue Jia - "Galectins Control mTOR in Response to Endomembrane Damage"

mTOR
AMPK
Autophagy
galectins
Subscribe to mTOR
Autophagy, Inflammation, & Metabolism Center of Biomedical Research Excellence

The AIM Center is funded by NIH grant P20GM121176, and it endows the state of New Mexico and the surrounding region with a state-of-the-art biomedical center and an intellectual and technological hub for cutting-edge research. On a larger scale, the AIM Center promises to be a nationally important center for the advancement of research on autophagy.

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