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lysosome

Galectin-3 Coordinates a Cellular System for Lysosomal Repair and Removal.

ESCRT
TFEB
endosome
galectins

Sustained activation of autophagy suppresses adipocyte maturation via a lipolysis-dependent mechanism.

Adipocyte maturation
mTORC1
Lipolysis
lysosome

Mammalian Atg8 proteins regulate lysosome and autolysosome biogenesis through SNAREs.

GABARAP
SNARE
TOR
lysosome
Abstract diagram

ATG5 provides host protection acting as a switch in the atg8ylation cascade between autophagy and secretion

Autophagy
Atg8ylation
lysosome
ATG5
Two new publications from the AIM Center

Another round of publications from The AIM Center!

AMPK
Metabolism
Autophagy
lysosome

Galectins Control mTOR in Response to Endomembrane Damage.

AMPK
APEX2
LC3
TAK1
Data Abstract

MITF-MIR211 axis is a novel autophagy amplifier system during cellular stress

Autophagy
cellular stress
lysosome
microRNA
Jingyue Gia - Abstract

Jingyue Jia - "Galectins Control mTOR in Response to Endomembrane Damage"

mTOR
AMPK
Autophagy
galectins
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Autophagy, Inflammation, & Metabolism Center of Biomedical Research Excellence

The AIM Center is funded by NIH grant P20GM121176, and it endows the state of New Mexico and the surrounding region with a state-of-the-art biomedical center and an intellectual and technological hub for cutting-edge research. On a larger scale, the AIM Center promises to be a nationally important center for the advancement of research on autophagy.

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